Anti-BCL2 therapy eliminates giant congenital melanocytic nevus by senolytic and immune induction

BCL2 expression and GCMN cell survival

BCL2 expression contributes to the prolonged survival of both P16+ senescent and Ki67+ proliferative giant congenital melanocytic nevus (GCMN) cells. Clinically, GCMN lesions maintain consistent dimensions…

Continue Reading